text_1: Increased microtubule acetylation repairs interference of axonal transport caused by LRRK2 Roc-COR domain mutations.
text_2: In vitro, increasing microtubule acetylation using deacetylase inhibitors or the tubulin acetylase αTAT1 prevents association of mutant LRRK2 with microtubules, and the deacetylase inhibitor trichostatin A (TSA) restores axonal transport.