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README.md

@@ -62,9 +62,11 @@ English
 
 ### Data Instances
 
+Records have the following structure
+
 ```json
-{"text": "The manifestations of acute intestinal obstruction depend on the nature of the underlying disease process, its location, and changes in blood flow (Fig. 355-1). Increased intestinal contractility, which occurs proximally and distal to the obstruction, is a characteristic response. Subsequently, intestinal peristalsis slows as the intestine or stomach proximal to the point of obstruction dilates and fills with gastrointestinal secretions and swallowed air. Although swallowed air is the primary contributor to intestinal distension, intraluminal air may also accumulate from fermentation, local carbon dioxide production, and altered gaseous diffusion.  Intraluminal dilation also increases intraluminal pressure. When luminal pressure exceeds venous pressure, venous and lymphatic drainage is impeded. Edema ensues, and the bowel wall proximal to the site of blockage may become hypoxemic. Epithelial necrosis can be identified within 12 h of obstruction. Ultimately, arterial blood supply may become so compromised that full-thickness ischemia, necrosis, and perforation result. Stasis increases the bacteria counts within the jejunum and ileum. The most commonly cultured intraluminal organisms are Escherichia coli, Streptococcus faecalis, and Klebsiella, which may also be recovered from mesenteric lymph nodes and other more distant sites.",
-  "source": "textbooks/en/InternalMed_Harrison.txt"}
+{"text": "The manifestations of acute intestinal obstruction depend on the nature of the underlying disease process, [..]", 
+ "source": "textbooks/en/InternalMed_Harrison.txt"}
 ```
 ## Dataset Creation